12% is too few lipid droplets.
lipodystrophy and cachexia.
I think it is technically wrong reversed, I think it should be the opposite, lipodystrophy and cachexia leads to too few lipid droplets.
None of the other options make any sense, the only plausible choice is proper development, differentiation and survival.
Lipodystrophy is the loss of failure to develop adequate amounts of adipose tissue for energy storage.
This is from my memory in the lecture, it is mutation in the AGPAT.
This is also from my memory in the lecture, it is AKT2 and nuclear receptor PPARy.
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